Metabolic alterations linked to T2D are well characterised by epidemiological and research primarily based studies. The pathogenesis and development of T2D is ascribed to four mechanisms; greater Sophisticated glycation finish merchandise (AGE) development, enhanced polyol pathway flux, activation of protein kinase C (PKC) isoforms, and improved hexosamine pathway flux[7]. Till https://www.directivepublications.org/journal-of-diabetology-research/